Associate Professor of Medicine, Johns Hopkins University School of Medicine
Hematology
Dr. Alison R. Moliterno, MD
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Dr. Alison R. Moliterno is an expert in the field of Hematology. He attended University at Buffalo, School of Medicine and Biomedical Sciences. He has 2 awards "Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Alison R. Moliterno has written publications. He has 40 publications published with PubMed. The lastest publication is: 'Impaired expression of the thrombopoietin receptor by platelets from patients with polycythemia vera.' He accepts Medicare payments and is listed with Medicare.gov.
Publications
- Polycythemia Vera: An Appraisal of the Biology and Management 10 Years After the Discovery of JAK2 V617F.
- Polycythemia vera Disease burden: contributing factors, impact on quality of life, and emerging treatment options.
- Back to Biology: New Insights on Inheritance in Myeloproliferative Disorders.
- Novel recurrent mutations in the RAS-like GTP-binding gene RIT1 in Myeloid malignancies.
- Multiple mechanisms deregulate EZH2 and histone H3 lysine 27 epigenetic changes in Myeloid malignancies.
- MicroRNA deregulation in Polycythemia vera and essential thrombocythemia patients.
- Could hypoxia increase the prevalence of thrombotic complications in Polycythemia vera?
- Age-related differences in Disease characteristics and clinical outcomes in polycythemia vera.
- Janus kinase inhibitors: an update on the progress and promise of targeted therapy in the Myeloproliferative neoplasms.
- Disruption of the ASXL1 gene is frequent in primary, post-essential thrombocytosis and post-Polycythemia vera myelofibrosis, but not essential thrombocytosis or polycy...
- AKNA: Another AT-hook transcription factor "hooking-up" with inflammation.
- Gender and Vascular Complications in the JAK2 V617F-Positive Myeloproliferative Neoplasms.
- Primary myelofibrosis and the Myeloproliferative neoplasms: the role of individual variation.
- Detection of exon 12 Mutations in the JAK2 gene: enhanced analytical sensitivity using clamped PCR and nucleotide sequencing.
- Acute cardiomyopathy in a patient with thrombotic thrombocytopenic purpura.
- Human-induced pluripotent stem cells from blood cells of healthy donors and patients with acquired blood Disorders.
- Hypersplenism induced by splenic Vein ligation.
- An absorbing problem.
- Molecular mimicry in the Chronic myeloproliferative disorders: reciprocity between quantitative JAK2 V617F and Mpl expression.
- Case 15-2006: the Budd-Chiari Syndrome and V617F mutation in JAK2.
- Platelet counts differ by sex, ethnicity, and age in the United States.
- Mpl Baltimore: a thrombopoietin receptor polymorphism associated with Thrombocytosis.
- Polymerized human Hb use in Acute chest syndrome: a case report.
- Biology and management of idiopathic myelofibrosis.
- A novel thrombopoietin signaling defect in Polycythemia vera platelets.
- Posttranslational processing of the thrombopoietin receptor is impaired in Polycythemia vera.
- Impaired expression of the thrombopoietin receptor by platelets from patients with Polycythemia vera.
- The quantitative JAK2 V617F neutrophil allele burden does not correlate with thrombotic risk in essential Thrombocytosis.
- Novel homo- and hemizygous mutations in EZH2 in Myeloid malignancies.
- Anemia of cancer.
- Disease burden at the progenitor level is a feature of primary myelofibrosis: a multivariable analysis of 164 JAK2 V617F-positive myeloproliferative neoplasm patients.
- Sex differences in the JAK2 V617F allele burden in Chronic myeloproliferative disorders.
- Phenotypic variability within the JAK2 V617F-positive MPD: roles of progenitor Cell and neutrophil allele burdens.
- Phenotypic variations and new mutations in JAK2 V617F-negative Polycythemia vera, erythrocytosis, and idiopathic myelofibrosis.
- Estimating platelet production in patients with HIV-related thrombocytopenia using the immature platelet fraction.
- Wiped out.
- Thrombotic and bleeding complications in four subpopulations of patients with essential thrombocythemia defined by c-Mpl protein expression and PRV-1 mRNA levels.
- Wasted sheep and premature infants: the role of trace metals in hematopoiesis.
- Clinical presentation and management of patients with circulating anticoagulants.
Schools
State University of New York At Buffalo
Johns Hopkins Bayview M C
Johns Hopkins University School Med
Procedures Preformed
- Plasmapheresis
Conditions Treated
- Acute Leukemia
- Anemia
- Antiphospholipid Syndrome (APS)
- Central Nervous System Lymphoma
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Doctors Specialties
- Hematology
Accepted Insurances
- Blue Card PPO
- Blue Preferred PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- CoreSource
- Coventry Health Care
- Coventry Health Care of Delaware PPO
- First Health (Coventry Health Care) PPO
- Geisinger Health Plan
- Golden Rule
- Choice Care PPO
- Medicaid
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Tricare
- Choice Plus POS
- Navigate HMO
- Options PPO
- United Healthcare
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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Johns Hopkins University -
Johns Hopkins Bayview Medical Center -
University at Buffalo, School of Medicine and Biomedical Sciences
Hospital
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Johns Hopkins Hospital -
Johns Hopkins Bayview Medical Center
Drug Facts
| NPI NUMBER | 1487604591 | |
| NPPES Provider LastName | MOLITERNO | |
| NPPES Provider FirstName | ALISON | |
| NPPES Provider ZIPCode | 212870005 | |
| NPPES Provider State | MD | |
| Specialty Description | Hematology | |
| Total Claim Count | 97.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1487604591 |
| Last Name Of The Provider | MOLITERNO |
| First Name Of The Provider | ALISON |
| View All |
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