Associate Professor, Medicine, University of Vermont College of Medicine
Internal Medicine
Dr. Benjamin T. Suratt, MD
Benjamin T. Suratt, MD is an expert in the field of Internal Medicine. He went to medical school at Columbia University College of Physicians & Surgeons. Columbia University College of Physicians & Surgeons is a top medical college. Columbia University College of Physicians & Surgeons has a rank of 8 in research and a 52 in primary care He over the years received 1 awards: "CMS Meaningful Use Stage 1 Certification". Dr. Benjamin T. Suratt is a published doctor and has 43 publications published. The lastest was: 'Airway epithelial NF-B activation promotes the ability to overcome inhalational antigen tolerance.' He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Airway epithelial NF-κB activation promotes the ability to overcome inhalational antigen tolerance.
- Obesity and Nutrition in Acute Respiratory Distress Syndrome.
- Active lifestyle: the next "smoking cessation"?
- Weight gain and lung Disease: the vagary of body mass index and the dilemma of the obese smoker.
- Glutaredoxin-1 Attenuates S-glutathionylation of the Death Receptor Fas and Decreases Resolution of Pseudomonas aeruginosa Pneumonia.
- The Role of Leptin in the Development of Pulmonary Neutrophilia in Infection and Acute Lung Injury.
- Effects of acute and Chronic low density lipoprotein exposure on neutrophil function.
- The weight of obesity on Lung health.
- Leptin as regulator of Pulmonary immune responses: Involvement in respiratory diseases.
- Interleukin-1 Receptor and Caspase-1 are Required for the Th17 Response in NO2-Promoted Allergic Airway Disease.
- Greasing the Way: The ABCs of HSPC Efflux from the Marrow.
- Obesity and Asthma: an inflammatory disease of adipose tissue not the airway.
- Essential role of IL-6 in protection against H1N1 influenza virus by promoting neutrophil survival in the Lung.
- Obesity is associated with neutrophil dysfunction and attenuation of murine acute Lung injury.
- Extreme Obesity and Outcomes in Critically Ill Patients.
- Serum Amyloid A Activates the NLRP3 Inflammasome and Promotes Th17 Allergic Asthma in Mice.
- Airway epithelial indoleamine 2,3-dioxygenase inhibits CD4+ T cells during Aspergillus fumigatus antigen exposure.
- Distinct functions of airway epithelial nuclear factor-kappaB activity regulate nitrogen dioxide-induced acute Lung injury.
- Dyslipidemia induces opposing effects on intraPulmonary and extraPulmonary host defense through divergent TLR response phenotypes.
- Slam haplotypes modulate the response to lipopolysaccharide in vivo through control of NKT cell number and function.
- Crosstalk between CXCR4/stromal derived factor-1 and VLA-4/VCAM-1 pathways regulates neutrophil retention in the bone marrow.
- Lower airway disease in Asthmatics with and without rhinitis.
- Derivation of Lung epithelium from human cord blood-derived mesenchymal stem cells.
- Pulmonary stromal-derived factor-1 expression and effect on neutrophil recruitment during acute lung injury.
- Novel therapies for the treatment of Cystic fibrosis: new developments in gene and stem cell therapy.
- Mechanisms of acute Lung injury/acute respiratory distress syndrome.
- Choosing the frequency of deep inflation in mice: balancing recruitment against ventilator-induced Lung injury.
- Limited restoration of Cystic fibrosis lung epithelium in vivo with adult bone marrow-derived cells.
- Acute Lung injury with endotoxin or NO2 does not enhance development of airway epithelium from bone marrow.
- Role of the CXCR4/SDF-1 chemokine axis in circulating neutrophil homeostasis.
- The in vitro production and characterization of neutrophils from embryonic stem cells.
- Human Pulmonary chimerism after hematopoietic stem cell transplantation.
- Selective suppression of neutrophil accumulation in ongoing Pulmonary inflammation by systemic inhibition of p38 mitogen-activated protein kinase.
- The Endogenous Th17 Response in NO2-Promoted Allergic Airway Disease Is Dispensable for Airway Hyperresponsiveness and Distinct from Th17 Adoptive Transfer.
- Plasma granulocyte colony-stimulating factor levels correlate with clinical outcomes in patients with acute Lung injury.
- Neutrophil maturation and activation determine anatomic site of clearance from circulation.
- A 48-year-old smoker with cough and weight loss.
- Role of p38 mitogen-activated protein kinase in a murine model of Pulmonary inflammation.
- Interferon-gamma for delayed Pulmonary toxicity syndrome resistant to steroids.
- The association between BMI and plasma cytokine levels in patients with acute Lung injury.
- NO2 inhalation induces maturation of Pulmonary CD11c+ cells that promote antigenspecific CD4+ T cell polarization.
- Relationship of adipokines with immune response and lung function in obese Asthmatic and non-Asthmatic women.
Schools
Columbia University College of Physicians And Surgeons
Massachusetts General Hospital
University Of Colorado Hospital
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Asthma
- Atelectasis
- Bronchiectasis
- View All
Doctors Specialties
- Critical Care Medicine
- Pulmonology
- Internal Medicine
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
-
University of Colorado -
Massachusetts General Hospital -
Columbia University College of Physicians & Surgeons
Drug Facts
| NPI NUMBER | 1922114164 | |
| NPPES Provider LastName | SURATT | |
| NPPES Provider FirstName | BENJAMIN | |
| NPPES Provider ZIPCode | 05401 | |
| NPPES Provider State | VT | |
| Specialty Description | Critical Care (Intensivists) | |
| Total Claim Count | 124.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1922114164 |
| Last Name Of The Provider | SURATT |
| First Name Of The Provider | BENJAMIN |
| View All |
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