Director, Cedars-Sinai Heart Institute, Los Angeles CA
Cardiology
Dr. Eduardo Marban, MD
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Eduardo Marban is an expert in the field of Cardiology. He went to Yale University School of Medicine. Yale University School of Medicine is a top medical university. Yale University School of Medicine has a rank of 7 in research and a 57 in primary care He received awards:"Fellow (FACC)". Doctor Eduardo Marban, MD is a published MD. He published 100 publications, including: 'Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse hearts.'
Publications
- Exosomes: Fundamental Biology and Roles in Cardiovascular Physiology.
- A corrole nanobiologic elicits tissue-activated MRI contrast enhancement and tumor-targeted toxicity.
- Wnt signalling suppresses voltage-dependent Na(+) channel expression in postnatal rat cardiomyocytes.
- Cellular postconditioning: allogeneic cardiosphere-derived cells reduce infarct size and attenuate microvascular obstruction when administered after reperfusion in pig...
- Endomyocardial biopsy technique for orthotopic heart transplantation and cardiac stem-cell harvesting.
- Engineered electrical conduction tract restores conduction in complete heart block: from in vitro to in vivo proof of concept.
- SHOX2 Overexpression Favors Differentiation of Embryonic Stem Cells into Cardiac Pacemaker Cells, Improving Biological Pacing Ability.
- Cardiospheres reverse adverse remodeling in chronic rat myocardial infarction: roles of soluble endoglin and Tgf-β signaling.
- Translating Stem Cell Research to Cardiac Disease Therapies: Pitfalls and Prospects for Improvement.
- Biological pacemaker created by minimally invasive somatic reprogramming in pigs with complete heart block.
- Therapeutic efficacy of cardiosphere-derived cells in a transgenic mouse model of non-ischaemic dilated cardiomyopathy.
- Exosomes as critical agents of cardiac regeneration triggered by cell therapy.
- Stimulation of endogenous cardioblasts by exogenous cell therapy after myocardial infarction.
- Importance of cell-cell contact in the therapeutic benefits of cardiosphere-derived cells.
- Cardiac BIN1 folds T-tubule membrane, controlling ion flux and limiting arrhythmia.
- Determination of Location, Size and Transmurality of Chronic Myocardial Infarction Without Exogenous Contrast Media Using Cardiac Magnetic Resonance Imaging at 3T.
- Human Cardiosphere-Derived Cells From Advanced Heart Failure Patients Exhibit Augmented Functional Potency in Myocardial Repair.
- Validation of contrast-enhanced magnetic resonance imaging to monitor regenerative efficacy after cell therapy in a porcine model of convalescent myocardial infarction.
- Moving Beyond Surrogate Endpoints in Cell Therapy Trials for Heart Disease.
- Intracoronary cardiosphere-derived cells after myocardial infarction: evidence for therapeutic regeneration in the final 1-year results of the CADUCEUS trial.
- Allogeneic cardiospheres safely boost cardiac function and attenuate adverse remodeling after myocardial infarction in immunologically mismatched rat strains.
- Intrinsic cardiac origin of human cardiosphere-derived cells.
- Heart to heart: Cardiospheres for myocardial regeneration.
- Kidney-derived stromal cells modulate dendritic and T cell responses.
- Genetic variations in nitric oxide synthase 1 adaptor protein are associated with sudden cardiac death in US white community-based populations.
- IK1 heterogeneity affects genesis and stability of spiral waves in cardiac myocyte monolayers.
- Ectopic expression of the sodium-iodide symporter enables imaging of transplanted cardiac stem cells in vivo by single-photon emission computed tomography or positron ...
- Durable Benefits of Cellular Postconditioning: Long-Term Effects of Allogeneic Cardiosphere-Derived Cells Infused After Reperfusion in Pigs with Acute Myocardial Infar...
- Allogeneic Cardiospheres Delivered via Percutaneous Transendocardial Injection Increase Viable Myocardium, Decrease Scar Size, and Attenuate Cardiac Dilatation in Porc...
- Relative Roles of CD90 and c-Kit to the Regenerative Efficacy of Cardiosphere-Derived Cells in Humans and in a Mouse Model of Myocardial Infarction.
- Magnetic antibody-linked nanomatchmakers for therapeutic cell targeting.
- Angiogenesis, cardiomyocyte proliferation and anti-fibrotic effects underlie structural preservation post-infarction by intramyocardially-injected cardiospheres.
- Magnetic enhancement of cell retention, engraftment, and functional benefit after intracoronary delivery of cardiac-derived stem cells in a rat model of ischemia/reper...
- Pharmacologically induced preconditioning with diazoxide: a novel approach to brain protection.
- Krüppel-like factor 4 (gut-enriched Krüppel-like factor) inhibits cell proliferation by blocking G1/S progression of the cell cycle.
- Voltage-dependent facilitation of cardiac L-type Ca channels expressed in HEK-293 cells requires beta-subunit.
- Virus-mediated modification of cellular excitability.
- Functional expression of exogenous proteins in mammalian sensory hair cells infected with adenoviral vectors.
- Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse hearts.
- Suppression of KATP currents by gene transfer of a dominant negative Kir6.2 construct.
- Acceleration of widespread adenoviral gene transfer to intact rabbit hearts by coronary perfusion with low calcium and serotonin.
- Mechanistic link between lidocaine block and inactivation probed by outer pore mutations in the rat micro1 skeletal muscle sodium channel.
- Mechanism for the cardioprotective effects of the calcium channel blocker clentiazem during ischemia and reperfusion.
- Mitochondrial ATP-dependent potassium channels: novel effectors of cardioprotection?
- Calcium and heart failure.
- Medical and cellular implications of stunning, hibernation, and preconditioning: an NHLBI workshop.
- Isoform-specific inhibition of L-type calcium channels by dihydropyridines is independent of isoform-specific gating properties.
- Mechanism of anode break stimulation in the heart.
- Structure and function of voltage-gated sodium channels.
- Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channel.
- Calcium cycling and contractile activation in intact mouse cardiac muscle.
- Subcellular metabolic transients and mitochondrial redox waves in heart cells.
- Suppression of neuronal and cardiac transient outward currents by viral gene transfer of dominant-negative Kv4.2 constructs.
- Priming effect of adenosine on K(ATP) currents in intact ventricular myocytes: implications for preconditioning.
- Direct inhibition of expressed cardiac L-type Ca2+ channels by S-nitrosothiol nitric oxide donors.
- Beat-to-beat QT interval variability: novel evidence for repolarization lability in ischemic and nonischemic dilated cardiomyopathy.
- Ca2+ 'sparks' and waves in intact ventricular muscle resolved by confocal imaging.
- Molecular motions within the pore of voltage-dependent sodium channels.
- Repolarization abnormalities, arrhythmia and sudden death in canine tachycardia-induced cardiomyopathy.
- Topology of the P segments in the sodium channel pore revealed by cysteine mutagenesis.
- Mechanisms of sodium/calcium selectivity in sodium channels probed by cysteine mutagenesis and sulfhydryl modification.
- Ultrarapid, highly efficient viral gene transfer to the heart.
- Ion channels as enzymes: analogy or homology?
- Differential modulation of cortical synaptic activity by calcineurin (phosphatase 2B) versus phosphatases 1 and 2A.
- Mechanism of Ca(2+)-dependent inactivation of L-type Ca2+ channels in GH3 cells: direct evidence against dephosphorylation by calcineurin.
- Proton inhibition of sodium channels: mechanism of gating shifts and reduced conductance.
- Role of troponin I proteolysis in the pathogenesis of stunned myocardium.
- Control of ion flux and selectivity by negatively charged residues in the outer mouth of rat sodium channels.
- Cell-type specificity of preconditioning in an in vitro model.
- Local anesthetics as effectors of allosteric gating. Lidocaine effects on inactivation-deficient rat skeletal muscle Na channels.
- The equine periodic paralysis Na+ channel mutation alters molecular transitions between the open and inactivated states.
- Selective effects of oxygen free radicals on excitation-contraction coupling in ventricular muscle. Implications for the mechanism of stunned myocardium.
- Reversal of potassium channel deficiency in cells from failing hearts by adenoviral gene transfer: a prototype for gene therapy for disorders of cardiac excitability a...
- Single-channel analysis of inactivation-defective rat skeletal muscle sodium channels containing the F1304Q mutation.
- External pore residue mediates slow inactivation in mu 1 rat skeletal muscle sodium channels.
- Coupling between fast and slow inactivation revealed by analysis of a point mutation (F1304Q) in mu 1 rat skeletal muscle sodium channels.
- Functional consequences of lidocaine binding to slow-inactivated sodium channels.
- Enhancement of ionic current and charge movement by coexpression of calcium channel beta 1A subunit with alpha 1C subunit in a human embryonic kidney cell line.
- Adjacent pore-lining residues within sodium channels identified by paired cysteine mutagenesis.
- Depth asymmetries of the pore-lining segments of the Na+ channel revealed by cysteine mutagenesis.
- Intrinsic myofilament alterations underlying the decreased contractility of stunned myocardium. A consequence of Ca2+-dependent proteolysis?
- Synergistic modulation of ATP-sensitive K+ currents by protein kinase C and adenosine. Implications for ischemic preconditioning.
- Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failure.
- Structure of the sodium channel pore revealed by serial cysteine mutagenesis.
- Prospects for genetic manipulation of cardiac excitability.
- Metabolic oscillations in heart cells.
- Stunned myocardium: a disease of the myofilaments?
- Background potassium current active during the plateau of the action potential in guinea pig ventricular myocytes.
- Role of sodium/calcium exchange in the mechanism of myocardial stunning: protective effect of reperfusion with high sodium solution.
- Relative roles of intracellular Ca2+ and pH in shaping myocardial contractile response to acute respiratory alkalosis.
- Regulation of intracellular calcium in cardiac muscle.
- Mechanism of the diastolic dysfunction induced by glycolytic inhibition. Does adenosine triphosphate derived from glycolysis play a favored role in cellular Ca2+ homeo...
- Myofilament Ca2+ sensitivity in intact versus skinned rat ventricular muscle.
- Mechanism of force inhibition by 2,3-butanedione monoxime in rat cardiac muscle: roles of [Ca2+]i and cross-bridge kinetics.
- Oscillations of membrane current and excitability driven by metabolic oscillations in heart cells.
- Disruption of intracellular Ca2+ homeostasis in hearts reperfused after prolonged episodes of ischemia.
- Control of steady-state intracellular pH in intact perfused ferret hearts.
- Sudden cardiac death in heart failure. The role of abnormal repolarization.
- Functional consequences of sulfhydryl modification in the pore-forming subunits of cardiovascular Ca2+ and Na+ channels.
Schools
Yale Univ Sch Of Med
Johns Hopkins Hospital
Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Fellow (FACC)
Education
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Johns Hopkins University -
Yale University School of Medicine
Hospital
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Cedars-Sinai Medical Center
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