Professor, Internal Medicine, Virginia Commonwealth University, Medical College of Virginia School of Medicine
Gastroenterology
Dr. John F. Kuemmerle, MD
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John Kuemmerle practices Gastroenterology. He went to Eastern Virginia Medical School. He has 2 awards "Regional Top Doctor" and "Fellow (AGAF)". John F. Kuemmerle, MD is a published doctor and has 60 publications published. The lastest was: 'IGFBP-3 and IGFBP-5 production by human intestinal muscle: reciprocal regulation by endogenous TGF-beta1.' John Kuemmerle accepts Medicare.
Publications
- Actin-Depolymerizing Factor and Cofilin-1 Have Unique and Overlapping Functions in Regulating Intestinal Epithelial Junctions and Mucosal Inflammation.
- Characterization of Genetic Loci That Affect Susceptibility to Inflammatory Bowel Diseases in African Americans.
- Noncanonical STAT3 Activation Regulates Excess TGF-β1 and Collagen I Expression in Muscle of Stricturing Crohn's Disease.
- Activation of the umami taste receptor (T1R1/T1R3) initiates the peristaltic reflex and pellet propulsion in the distal colon.
- LOSS OF γ-CYTOPLASMIC ACTIN TRIGGERS MYOFIBROBLAST TRANSITION OF HUMAN EPITHELIAL CELLS.
- ABIM Maintenance of Certification 2014: Navigating the Challenges to Find Opportunities for Success.
- Jun kinase-induced overexpression of leukemia-associated Rho GEF (LARG) mediates sustained hypercontraction of longitudinal smooth muscle in inflammation.
- Cytokine-induced iNOS and ERK1/2 inhibit AC5/6 activity and stimulate PDE4D5 activity in Intestinal longitudinal smooth muscle.
- Mechanisms That Mediate the Development of Fibrosis in Patients With Crohn's Disease.
- Hypercontractility of Intestinal Longitudinal Smooth Muscle Induced by Cytokines is Mediated by NFκB/AMP-Activated Kinase/MLC Kinase Pathway.
- Regulation of Gβγi-Dependent PLC-β3 Activity in Smooth Muscle: Inhibitory Phosphorylation of PLC-β3 by PKA and PKG and Stimulatory Phosphorylation of Gαi-GTPase-Activa...
- Increased Activation of Latent TGF-β1 by αVβ3 in Human Crohn's Disease and Fibrosis in TNBS Colitis Can Be Prevented by Cilengitide.
- Effective use of technology in gastroenterology training.
- Insulin-like growth factors in the gastroIntestinal tract and liver.
- Amelioration of excess collagen IαI, fibrosis, and smooth muscle growth in TNBS-induced colitis in IGF-I(+/-) mice.
- Endogenous IGFBP-3 regulates excess collagen expression in intestinal smooth muscle cells of Crohn's Disease strictures.
- Endogenous IGF-I and alphaVbeta3 integrin ligands regulate increased smooth muscle hyperplasia in stricturing Crohn's Disease.
- Insulin-like growth factor-binding protein-5 stimulates growth of human Intestinal muscle cells by activation of G{alpha}i3.
- Report of the multisociety task force on GI training.
- Modulation of motor and sensory pathways of the peristaltic reflex by cannabinoids.
- Endogenous IGF-I and alpha v beta3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis.
- Occupation of alphavbeta3-integrin by endogenous ligands modulates IGF-I receptor activation and proliferation of human Intestinal smooth muscle.
- A 14-kDa cathepsin L-derived carboxyl IGFBP-2 fragment is sequestered by cultured rat ileal crypt cells.
- Endogenous IGF-I protects human Intestinal smooth muscle cells from apoptosis by regulation of GSK-3 beta activity.
- IGFBP-3 activates TGF-beta receptors and directly inhibits growth in human Intestinal smooth muscle cells.
- IGF-I stimulates human Intestinal smooth muscle cell growth by regulation of G1 phase cell cycle proteins.
- IGF-I elicits growth of human Intestinal smooth muscle cells by activation of PI3K, PDK-1, and p70S6 kinase.
- Insulin-like growth factor-binding protein-5 (IGFBP-5) stimulates growth and IGF-I secretion in human Intestinal smooth muscle by Ras-dependent activation of p38 MAP k...
- Diagnostic endoscopy via gastrostomy or PEG stoma.
- Feedback regulation of intracellular calcium release.
- Therapy of gastroIntestinal motility disorders: moving in the right direction.
- Serotonin receptor blockade improves cardiac output and hypoxia in porcine ARDS.
- Serotonin release is mediated by muscarinic receptors on Duodenal mucosal cells.
- Motility disorders of the small intestine: new insights into old problems.
- Beta-adrenoceptors on Duodenal mucosal cells mediate venous serotonin release.
- GastroIntestinal hormone responses to meals before and after gastric bypass and vertical banded gastroplasty.
- Serotonin neural receptors mediate motilin-induced motility in isolated, vascularly perfused canine jejunum.
- Agonist-stimulated cyclic ADP ribose. Endogenous modulator of Ca(2+)-induced Ca2+ release in Intestinal longitudinal muscle.
- Characterization of opioid receptors in Intestinal muscle cells by selective radioligands and receptor protection.
- Synergistic regulation of NOS II expression by IL-1 beta and TNF-alpha in cultured rat colonic smooth muscle cells.
- Endogenous IGF-I regulates IGF binding protein production in human Intestinal smooth muscle cells.
- 5-HT released by mucosal stimuli initiates peristalsis by activating 5-HT4/5-HT1p receptors on sensory CGRP neurons.
- Agonist-activated, ryanodine-sensitive, IP3-insensitive Ca2+ release channels in longitudinal muscle of intestine.
- IGF-I stimulates Intestinal muscle cell growth by activating distinct PI 3-kinase and MAP kinase pathways.
- IGFBP-3 and IGFBP-5 production by human Intestinal muscle: reciprocal regulation by endogenous TGF-beta1.
- Coexistence of histamine H1 and H2 receptors coupled to distinct signal transduction pathways in isolated Intestinal muscle cells.
- Autocrine regulation of growth in cultured human Intestinal muscle by growth factors.
- Longitudinal smooth muscle of the mammalian intestine. A model for Ca2+ signaling by cADPR.
- Agonist-mediated activation of PLA2 initiates Ca2+ mobilization in Intestinal longitudinal smooth muscle.
- Expression of endothelial nitric oxide synthase in human and rabbit gastroIntestinal smooth muscle cells.
- Differential changes in brain-derived neurotrophic factor and extracellular signal-regulated kinase in rat primary afferent pathways with colitis.
- Augmentation of the ascending component of the peristaltic reflex and substance P release by glial cell line-derived neurotrophic factor.
- Selective expression of vasoactive Intestinal peptide (VIP)2/pituitary adenylate cyclase-activating polypeptide (PACAP)3 receptors in rabbit and guinea pig gastric and...
- Regulation of IGFBP-4 levels in human Intestinal muscle by an IGF-I-activated, confluence-dependent protease.
- Coexistence of contractile and relaxant 5-hydroxytryptamine receptors coupled to distinct signaling pathways in Intestinal muscle cells: convergence of the pathways on...
- Coexpression of 5-HT2A and 5-HT4 receptors coupled to distinct signaling pathways in human Intestinal muscle cells.
- Distinct 5-HT receptors mediate the peristaltic reflex induced by mucosal stimuli in human and guinea pig intestine.
- Sustained muscle contraction induced by agonists, growth factors, and Ca(2+) mediated by distinct PKC isozymes.
- Coupling of the insulin-like growth factor-I receptor tyrosine kinase to Gi2 in human Intestinal smooth muscle: Gbetagamma -dependent mitogen-activated protein kinase ...
Schools
Eastern Virginia Medical School Of The Medical College Of Hampton Roads
Med College Va
Procedures Preformed
- Colonoscopy, Proctosigmoidoscopy, and Sigmoidoscopy
- Endoscopy (Esophagus, Stomach, Small Intestine)
- Manometry
Conditions Treated
- Abdominal Pain
- Anemia
- Benign Neoplasm of the Digestive System
- Biliary Atresia
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Doctors Specialties
- Gastroenterology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Pathway X Tier Hosp HealthKeepers DirectAccess VA
- HealthKeepers HMO
- KeyCare PPO
- Assurant Health
- Blue Card PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- Southern Health PPO
- Coventry Health Care of Virginia HMO
- Choice Care PPO
- Mail Handlers Benefit Plan (MHBP)
- MedHealthInsurance
- Medicare MCR
- Meritain Health
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
- Wells Fargo Insurance
Awards
- Regional Top Doctor
- Fellow (AGAF)
Education
-
Virginia Commonwealth University Health System -
Eastern Virginia Medical School
Drug Facts
| NPI NUMBER | 1356431902 | |
| NPPES Provider LastName | KUEMMERLE | |
| NPPES Provider FirstName | JOHN | |
| NPPES Provider ZIPCode | 232985051 | |
| NPPES Provider State | VA | |
| Specialty Description | Gastroenterology | |
| Total Claim Count | 85.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1356431902 |
| Last Name Of The Provider | KUEMMERLE |
| First Name Of The Provider | JOHN |
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