Professor of Medicine, Massachusetts General Hospital
Endocrinology
Dr. Marie B. Demay, MD
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Marie Demay is an expert in the field of Endocrinology. He researched medicine at University of Saskatchewan College of Medicine. He has 2 awards "Regional Top Doctor" and "Elected Member". Marie B. Demay, MD is a published doctor. He has 67 publications published. The most current publication "Normalization of mineral ion homeostasis by dietary means prevents hyperparathyroidism, rickets, and osteomalacia, but not alopecia in vitamin D receptor-ablated mice." Marie Demay accepts Medicare.
Publications
- Phosphate Interacts With PTHrP to Regulate Endochondral Bone Formation.
- The Vitamin D receptor is required for activation of cWnt and Hedgehog signaling in keratinocytes.
- Effect of Bisphosphonates on the Rapidly Growing Male Murine Skeleton.
- Ligand-Dependent Actions of the Vitamin D Receptor Are Required for Activation of TGF-ò Signaling during the Inflammatory Response to Cutaneous Injury.
- Physiological Insights from the Vitamin D Receptor Knockout Mouse.
- The hair cycle and Vitamin D receptor.
- Lymphoid enhancer-binding factor-1 (LEF1) interacts with the DNA binding domain of the Vitamin D receptor.
- The receptor-dependent actions of 1,25-dihydroxyVitamin D are required for normal growth plate maturation in NPt2a knockout mice.
- The biology and pathology of Vitamin D control in bone.
- Phosphate-induced apoptosis of hypertrophic chondrocytes is associated with a decrease in mitochondrial membrane potential and is dependent upon Erk1/2 phosphorylation.
- Acute phosphate restriction leads to impaired fracture healing and resistance to BMP-2.
- Phosphate regulates embryonic endochondral bone development.
- Perichondrial expression of Wdr5 regulates chondrocyte proliferation and differentiation.
- Impaired bone development and increased mesenchymal progenitor cells in calvaria of RB1-/- mice.
- Case records of the Massachusetts General Hospital. Case 16-2008. A 46-year-old woman with bone pain.
- Wdr5 is essential for osteoblast differentiation.
- Vitamin D receptor is essential for normal keratinocyte stem cell function.
- VDR-mediated inhibition of DKK1 and SFRP2 suppresses adipogenic differentiation of murine bone marrow stromal cells.
- Calcium and vitamin D: what is known about the effects on growing bone.
- Wdr5, a WD-40 protein, regulates osteoblast differentiation during embryonic bone development.
- Mechanism of Vitamin D receptor action.
- Hypophosphatemia leads to Rickets by impairing caspase-mediated apoptosis of hypertrophic chondrocytes.
- Genetic dissection of phosphate- and Vitamin D-mediated regulation of circulating Fgf23 concentrations.
- Ligand-independent actions of the Vitamin D receptor maintain hair follicle homeostasis.
- The effects of BIG-3 on osteoblast differentiation are not dependent upon endogenously produced BMPs.
- Altered pharmacokinetics of 1alpha,25-dihydroxyVitamin D3 and 25-hydroxyVitamin D3 in the blood and tissues of the 25-hydroxyVitamin D-24-hydroxylase (Cyp24a1) null mo...
- Osteoblasts lacking the Vitamin D receptor display enhanced osteogenic potential in vitro.
- BIG-3, a novel WD-40 repeat protein, is expressed in the developing growth plate and accelerates chondrocyte differentiation in vitro.
- Rickets in VDR null mice is secondary to decreased apoptosis of hypertrophic chondrocytes.
- Transcriptional repression of the rat osteocalcin gene by deltaEF1.
- Muscle: a nontraditional 1,25-dihydroxyVitamin D target tissue exhibiting classic hormone-dependent Vitamin D receptor actions.
- [Opportunistic screening of breast Cancer in France].
- [Familial Amanita phalloides poisoning].
- [Pneumococcal peritonitis].
- Comparison of renal and osseous binding of paraThyroid hormone and hormonal fragments.
- Nucleotide sequence of cloned cDNAs encoding chicken preproparaThyroid hormone.
- The 88th Annual Meeting of the Endocrine Society, June 24-27, 2006, Boston MA, USA: selected pediatric presentations.
- Vitamin D and human health: lessons from Vitamin D receptor null mice.
- The Vitamin D receptor, the skin and stem cells.
- Role of the Vitamin D receptor in hair follicle biology.
- Targeting expression of the human Vitamin D receptor to the keratinocytes of Vitamin D receptor null mice prevents alopecia.
- Cloning and characterization of a novel WD-40 repeat protein that dramatically accelerates osteoblastic differentiation.
- Cloning and characterization of a novel protein kinase that impairs osteoblast differentiation in vitro.
- Fibromodulin is expressed by both chondrocytes and osteoblasts during fetal bone development.
- Metabolic and cellular analysis of alopecia in Vitamin D receptor knockout mice.
- TBX1 is responsible for cardiovascular defects in velo-cardio-facial/DiGeorge Syndrome.
- Vitamin D deficiency and disorders of vitamin D Metabolism.
- BMP-2 induces the expression of activin betaA and follistatin in vitro.
- Deficient mineralization of intramembranous bone in Vitamin D-24-hydroxylase-ablated mice is due to elevated 1,25-dihydroxyVitamin D and not to the absence of 24,25-di...
- Evaluation of keratinocyte proliferation and differentiation in Vitamin D receptor knockout mice.
- Rescue of the skeletal phenoType of vitamin D receptor-ablated mice in the setting of normal mineral ion homeostasis: formal histomorphometric and biomechanical analyses.
- Transcriptional repression of the rat osteocalcin gene: role of two intronic CCTCCT motifs.
- Characterization of an enhancer required for 1,25-dihydroxyVitamin D3-dependent transactivation of the rat osteocalcin gene.
- Normalization of mineral ion homeostasis by dietary means prevents hyperparaThyroidism, rickets, and osteomalacia, but not alopecia in vitamin D receptor-ablated mice.
- Analysis of vitamin D-dependent Calcium-binding protein messenger ribonucleic acid expression in mice lacking the vitamin D receptor.
- Regions of the rat osteocalcin gene which mediate the effect of 1,25-dihydroxyVitamin D3 on gene transcription.
- DNA sequences in the rat osteocalcin gene that bind the 1,25-dihydroxyVitamin D3 receptor and confer responsiveness to 1,25-dihydroxyVitamin D3.
- Sequences in the human paraThyroid hormone gene that bind the 1,25-dihydroxyvitamin D3 receptor and mediate transcriptional repression in response to 1,25-dihydroxyvit...
- Characterization of 1,25-dihydroxyVitamin D3 receptor interactions with target sequences in the rat osteocalcin gene.
- Targeted ablation of the vitamin D receptor: an animal model of vitamin D-dependent rickets Type II with alopecia.
- Cloning and characterization of the Vitamin D receptor from Xenopus laevis.
- DNA sequences downstream from the Vitamin D response element of the rat osteocalcin gene are required for ligand-dependent transactivation.
- Vitamin D receptor binding to the negative human paraThyroid hormone vitamin D response element does not require the retinoid x receptor.
- Identification of an osteoblastic silencer element in the first intron of the rat osteocalcin gene.
- A single up-stream element confers responsiveness to 1,25-dihydroxyVitamin D3 and tumor necrosis factor-alpha in the rat osteocalcin gene.
- Wdr5, a novel WD repeat protein, regulates osteoblast and chondrocyte differentiation in vivo.
Schools
University of Saskatchewan College of Medicine
Plains Health Center
Royal Victoria Hospital
Conditions Treated
- Abnormal Thyroid
- Adrenal Incidentaloma
- Adrenal Insufficiency
- Calcium Metabolism Disorders
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Doctors Specialties
- Endocrinology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- BlueConnect HMO
- Blue with Managed Care Behavioral Health HMO
- Preferred Blue PPO
- Cigna HMO
- Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- GHI PPO
- Fallon Community Health Plan
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Network Health
- Tufts PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Awards
- Regional Top Doctor
- Elected Member
Education
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Massachusetts General Hospital -
University of Saskatchewan College of Medicine
Hospital
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Massachusetts General Hospital
Drug Facts
| NPI NUMBER | 1528058153 | |
| NPPES Provider LastName | DEMAY | |
| NPPES Provider FirstName | MARIE | |
| NPPES Provider ZIPCode | 021142621 | |
| NPPES Provider State | MA | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 91.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1528058153 |
| Last Name Of The Provider | DEMAY |
| First Name Of The Provider | MARIE |
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