Physician
Cardiology
Dr. Martin M. Lewinter, MD
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Martin Lewinter works in the field of Cardiology. He went to NYU School of Medicine. He received awards:"CMS Meaningful Use Stage 1 Certification" and "Fellow (FACC)". Martin M. Lewinter is also published. He has 52 publications published. The lastest publication: "Relationship of exercise capacity and left ventricular dimensions in patients with a normal ejection fraction. An exploratory study.' He is registered with Medicare.
Publications
- Nitrate's effect on activity tolerance in Heart failure with preserved ejection fraction trial: rationale and design.
- Myocardial Stiffness in Patients with Heart Failure and a Preserved Ejection Fraction: Contributions of Collagen and Titin.
- Resting Ventricular-Vascular Function and Exercise Capacity in Heart Failure With Preserved Ejection Fraction: A RELAX Trial Ancillary Study.
- Metabolic Syndrome, C-reactive Protein, and Mortality in U.S. Blacks and Whites: The Reasons for Geographic and Racial Differences in Stroke (REGARDS) Study.
- Esophageal assessments of left ventricular filling pressures: A proof-of-concept study.
- Low-Dose Dopamine or Low-Dose Nesiritide in Acute Heart Failure With Renal Dysfunction: The ROSE Acute Heart Failure Randomized Trial.
- Mechanisms of Diastolic Dysfunction in Heart Failure With a Preserved Ejection Fraction: If It's Not One Thing It's Another.
- Cardiac Titin and Heart Disease.
- Effect of phosphodiesterase-5 inhibition on exercise capacity and clinical status in Heart failure with preserved ejection fraction: a randomized clinical trial.
- Titin is a major human Disease gene.
- High dose intraCoronary N-acetylcysteine in a porcine model of ST-elevation myocardial infarction.
- Elevated rates of force development and MgATP binding in F764L and S532P myosin mutations causing dilated Cardiomyopathy.
- Cardiac myosin isoforms exhibit differential rates of MgADP release and MgATP binding detected by Myocardial viscoelasticity.
- Resistance training alters skeletal muscle structure and function in human Heart failure: effects at the tissue cellular and molecular levels.
- Erythropoietin induces positive inotropic and lusitropic effects in murine and human myocardium.
- Tachycardia-induced diastolic dysfunction and resting tone in myocardium from patients with a normal ejection fraction.
- Chronic Heart failure reduces Akt phosphorylation in human skeletal muscle: relationship to muscle size and function.
- Effect of Resistance Training on Physical Disability in Chronic Heart Failure.
- Research priorities in hypertrophic cardiomyopathy: report of a Working Group of the National Heart, Lung, and Blood Institute.
- Cardiac titin: a multifunctional giant.
- Extensible behavior of titin in the miniswine left ventricle.
- Mechanisms underlying skeletal muscle weakness in human Heart failure: alterations in single fiber myosin protein content and function.
- Effects of cardiac myosin isoform variation on myofilament function and crossbridge kinetics in transgenic rabbits.
- N-acetylcysteine-enhanced contrast provides cardiorenal protection.
- Metabolic syndrome, inflammation, and incident Heart failure in the elderly: the cardiovascular health study.
- Quantification of protein phosphorylation by liquid chromatography-mass spectrometry.
- Genes overexpressed in cerebral arteries following salt-induced hypertensive Disease are regulated by angiotensin II, JunB, and CREB.
- Left ventricular end-diastolic volume is normal in patients with Heart failure and a normal ejection fraction: a renewed consensus in diastolic Heart failure.
- Coronary venous capture of contrast during angiography.
- Intracellular distributions of essential elements in cardiomyocytes.
- wnt3a but not wnt11 supports self-renewal of embryonic stem cells.
- Skeletal muscle myofibrillar mRNA expression in Heart failure: relationship to local and circulating hormones.
- Cardiomyocyte function associated with hyperactivity and/or Hypertension in genetic models of LV hypertrophy.
- Sarcomeric proteins in hypertrophied and failing myocardium: an overview.
- Functional consequences of sarcomeric protein abnormalities in failing myocardium.
- Regional differences in the force-frequency relation of human left ventricular myocardium in Mitral regurgitation: implications for ventricular shape.
- Skeletal muscle myofibrillar protein metabolism in Heart failure: relationship to immune activation and functional capacity.
- Left ventricular form and function: scientific priorities and strategic planning for development of new views of Disease.
- Thin-filament-based modulation of contractile performance in human Heart failure.
- Titin isoforms in Heart failure: are there benefits to supersizing?
- Effect of cardiac myosin binding protein-C on mechanoenergetics in mouse myocardium.
- Increased atrial and brain natriuretic peptides in adults with cyanotic congenital Heart disease: enhanced understanding of the relationship between hypoxia and natriu...
- Endothelin receptor blockade has an oxygen-saving effect in Dahl salt-sensitive rats with Heart failure.
- Effect of Heart failure on the regulation of skeletal muscle protein synthesis, breakdown, and apoptosis.
- Altered myocardial thin-filament function in the failing Dahl salt-sensitive rat Heart: amelioration by endothelin blockade.
- Pericardial Diseases.
- Titin: an endosarcomeric protein that modulates Myocardial stiffness in DCM.
- Myofilament remodeling during the progression of Heart failure.
- Changes in titin isoform expression in pacing-induced cardiac Failure give rise to increased passive muscle stiffness.
- Cardiac troponin T isoforms demonstrate similar effects on mechanical performance in a regulated contractile system.
- Relationship of exercise capacity and left ventricular dimensions in patients with a normal ejection fraction. An exploratory study.
Schools
New York University
Bellevue Hospital Center
Uc San Diego Med Center
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac MRI (Magnetic Resonance Imaging) of Heart or Chest
- Cardiac Myocardial Perfusion Imaging
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Conditions Treated
- Aneurysm and Dissection of Heart
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Ectasia
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Doctors Specialties
- Cardiology
Accepted Insurances
- Choice Plus POS II
- Blue Card PPO
- Capital District Physician's Health Plan (CDPHP) HMO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- CoreSource
- First Health (Coventry Health Care) PPO
- Medicare Advantage PFFS
- Humana
- Medicaid
- Medicare MCR
- POMCO Group
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- Universal American
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACC)
Education
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NYU School of Medicine
Drug Facts
| NPI NUMBER | 1578652996 | |
| NPPES Provider LastName | LEWINTER | |
| NPPES Provider FirstName | MARTIN | |
| NPPES Provider ZIPCode | 054034407 | |
| NPPES Provider State | VT | |
| Specialty Description | Cardiology | |
| Total Claim Count | 362.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1578652996 |
| Last Name Of The Provider | LEWINTER |
| First Name Of The Provider | MARTIN |
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