Professor, Internal Medicine, University of Iowa College of Medicine
Cardiology
Dr. Robert B. Felder, MD
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Robert Felder is an expert in the field of Cardiology. He graduated from University of North Carolina at Chapel Hill School of Medicine. University of North Carolina at Chapel Hill School of Medicine is a top medical university. University of North Carolina at Chapel Hill School of Medicine has a rank of 22 in research and a 2 in primary care Robert Felder is also a published doctor. He has 39 paper published. The most recent paper is: Central Renin-Angiotensin System Activation and Inflammation Induced by High-Fat Diet Sensitize Angiotensin II-Elicited Hypertension.
Publications
- Central Renin-Angiotensin System Activation and Inflammation Induced by High-Fat Diet Sensitize Angiotensin II-Elicited Hypertension.
- Inhibition of Brain Mitogen-Activated Protein Kinase Signaling Reduces Central Endoplasmic Reticulum Stress and Inflammation and Sympathetic Nerve Activity in Heart Fa...
- Proinflammatory Cytokines Upregulate Sympathoexcitatory Mechanisms in the Subfornical Organ of the Rat.
- Central SDF-1/CXCL12 Expression and Its Cardiovascular and Sympathetic Effects: The Role of Angiotensin II, TNF-α and MAPK Signaling.
- Subfornical Organ Mediates Sympathetic and Hemodynamic Responses to Blood-Borne Proinflammatory Cytokines.
- Early Interference With p44/42 Mitogen-Activated Protein Kinase Signaling in Hypothalamic Paraventricular Nucleus Attenuates Angiotensin II-Induced Hypertension.
- Differential regulation of EHD3 in human and mammalian Heart failure.
- Central Actions of the Chemokine Stromal Cell-Derived Factor 1 Contribute to Neurohumoral Excitation in Heart Failure Rats.
- Peroxisome Proliferator-Activated Receptor-? Regulates Inflammation and Renin-Angiotensin System Activity in the Hypothalamic Paraventricular Nucleus and Ameliorates P...
- Aldosterone-Induced Brain MAPK Signaling and Sympathetic Excitation Are Angiotensin II Type-1 Receptor Dependent.
- EP3 Receptors Mediate PGE2-Induced Hypothalamic Paraventricular Nucleus Excitation and Sympathetic Activation.
- Brain perivascular macrophages and the sympathetic response to inflammation in rats after myocardial infarction.
- Autonomic cardiovascular modulation.
- Angiotensin II upregulates hypothalamic AT1 receptor expression in rats via the mitogen-activated protein kinase pathway.
- Mitogen-activated protein kinases mediate upregulation of hypothalamic angiotensin II type 1 receptors in Heart failure rats.
- Angiotensin II-triggered p44/42 mitogen-activated protein kinase mediates sympathetic excitation in Heart failure rats.
- Inhibition of brain proinflammatory cytokine synthesis reduces hypothalamic excitation in rats with ischemia-induced Heart failure.
- Does aldosterone upregulate the brain renin-angiotensin system in rats with Heart failure?
- Aldosterone acts centrally to increase brain renin-angiotensin system activity and oxidative stress in normal rats.
- Central gene transfer of interleukin-10 reduces hypothalamic inflammation and evidence of Heart failure in rats after myocardial infarction.
- Increased cyclooxygenase-2 expression in hypothalamic paraventricular nucleus in rats with Heart failure: role of nuclear factor kappaB.
- Novel effect of mineralocorticoid receptor antagonism to reduce proinflammatory cytokines and hypothalamic activation in rats with ischemia-induced Heart failure.
- 11beta-hydroxysteroid dehydrogenase type 2 activity in hypothalamic paraventricular nucleus modulates sympathetic excitation.
- Neuroendocrine and cytokine profile of chronic mild stress-induced anhedonia.
- Brain angiotensin-converting enzyme activity and autonomic regulation in Heart failure.
- Neural regulation of the proinflammatory cytokine response to acute myocardial infarction.
- Acute myocardial infarction induces hypothalamic cytokine synthesis.
- Melanocortin receptors mediate the excitatory effects of blood-borne murine leptin on hypothalamic paraventricular neurons in rat.
- Increased susceptibility to ventricular Arrhythmias in a rodent model of experimental depression.
- Mineralocorticoids act centrally to regulate blood-borne tumor necrosis factor-alpha in normal rats.
- Cardiovascular and renal sympathetic activation by blood-borne TNF-alpha in rat: the role of central prostaglandins.
- Cytokine mediation of experimental Heart failure-induced anhedonia.
- Heart failure and the brain: new perspectives.
- Central mineralocorticoid receptor blockade decreases plasma TNF-alpha after Coronary artery ligation in rats.
- The renin-angiotensin-aldosterone system excites hypothalamic paraventricular nucleus neurons in Heart failure.
- alpha(2)-Adrenergic receptors in NTS facilitate baroreflex function in adult spontaneously hypertensive rats.
- Forebrain-mediated adaptations to myocardial infarction in the rat.
- Forebrain renin-angiotensin system has a tonic excitatory influence on renal sympathetic nerve activity.
Schools
University of North Carolina At Chapel Hill
Duke University Med Center
University Iowa Hosps
Conditions Treated
- Aortic Aneurysm
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
- Cardiomyopathy
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Doctors Specialties
- Cardiology
Accepted Insurances
- Choice Plus POS II
- Great West Healthcare-Cigna PPO
- Health Alliance PPO
Education
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University of Iowa Hospitals and Clinics -
Duke University Hospital -
University of North Carolina at Chapel Hill School of Medicine
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