Professor, Dermatology, University of California, Davis, School of Medicine

Dermatology

Roslyn Isseroff practices Dermatology. She researched medicine at NYU School of Medicine. Roslyn Isseroff is a published doctor. She published 50 publications, including: 'Hypoxic pre-conditioning increases the infiltration of endothelial cells into scaffolds for dermal regeneration pre-seeded with mesenchymal stem cells.'

Publications

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• Hypoxic Preconditioning of Mesenchymal Stromal Cells Induces Metabolic Changes, Enhances Survival and Promotes Cell Retention in Vivo.
• Importance of defining experimental conditions in a mouse excisional wound model.
• Recovery and cultivation of keratinocytes from shipped mouse Skin.
• Diabetic Foot Ulcer: An Evidence-Based Treatment Update.
• Full-thickness splinted Skin wound healing models in db/db and heterozygous mice: Implications for wound healing impairment.
• Crosstalk Between Adrenergic and Toll-Like Receptors in Human Mesenchymal Stem Cells and Keratinocytes: A Recipe for Impaired Wound Healing.
• Acute Wounding Alters the Beta2-Adrenergic Signaling and Catecholamine Synthetic Pathways in Keratinocytes.
• Epidermal Growth Factor-Functionalized Polymeric Multilayer Films: Interplay Between Spatial Location and Bioavailability of EGF.
• Successful treatment of a chronic venous leg ulcer using a topical beta-blocker.
• Catecholamine Stress Alters Neutrophil Trafficking and Impairs Wound Healing by β2 Adrenergic Receptor Mediated Upregulation of IL-6.
• Cellular versus acellular matrix devices in treatment of diabetic Foot ulcers: study protocol for a comparative efficacy randomized controlled trial.
• Galectin-3 Regulates IntraCellular Trafficking of EGFR through Alix and Promotes Keratinocyte Migration.
• β2AR Antagonists and β2AR Gene Deletion Both Promote Skin Wound Repair Processes.
• Electrical Activation of Wound-Healing Pathways.
• Label-retaining Cells of the bladder: candidate urothelial stem Cells.
• Beta1 integrin expression pattern in transitional urothelium does not allow for efficient stem Cell enrichment as in other epithelia.
• Rat urothelium: improved techniques for serial cultivation, expansion, freezing and reconstitution onto aCellular matrix.
• Toll-like receptors and diabetes: a therapeutic perspective.
• Di-rhamnolipid from Pseudomonas aeruginosa displays differential effects on human keratinocyte and fibroblast cultures.
• Keratinocyte proximity and contact can play a significant role in determining mesenchymal stem Cell fate in human tissue.
• Beta2-adrenergic receptor activation delays wound healing.
• Beta-adrenergic receptor agonists delay while antagonists accelerate epithelial wound healing: evidence of an endogenous adrenergic network within the corneal epithelium.
• beta4 integrin and epidermal growth factor coordinately regulate electric field-mediated directional migration via Rac1.
• Cyclic AMP mediates keratinocyte directional migration in an electric field.
• Wound re-epithelialization: modulating keratinocyte migration in wound healing.
• PP2A activation by beta2-adrenergic receptor agonists: novel regulatory mechanism of keratinocyte migration.
• Direct binding of integrin alphavbeta3 to FGF1 plays a role in FGF1 signaling.
• Beta adrenergic receptors in keratinocytes.
• Responses of the 27-kDa heat shock protein to UVB irradiation in human epidermal melanocytes.
• The linear excisional wound: an improved model for human ex vivo wound epithelialization studies.
• Human dermal fibroblasts do not exhibit directional migration on collagen I in direct-current electric fields of physiological strength.
• Heat shock protein 27 is expressed in normal and malignant human melanocytes in vivo.
• UVB irradiation-induced changes in the 27-kd heat shock protein (HSP27) in human corneal epithelial Cells.
• The beta 2-adrenergic receptor activates pro-migratory and pro-proliferative pathways in dermal fibroblasts via divergent mechanisms.
• beta-Adrenergic receptor antagonists accelerate Skin wound healing: evidence for a catecholamine synthesis network in the epidermis.
• Topical negative pressure devices: use for enhancement of healing chronic wounds.
• Electrical stimulation of wound healing.
• Arsenite pre-conditioning reduces UVB-induced apoptosis in corneal epithelial Cells through the anti-apoptotic activity of 27 kDa heat shock protein (HSP27).
• Does inflammation have a role in the pathogenesis of venous Ulcers? A critical review of the evidence.
• Dynamics of neutrophil infiltration during cutaneous wound healing and Infection using fluorescence imaging.
• Stress-mediated increases in systemic and local epinephrine impair Skin wound healing: potential new indication for beta blockers.
• Power line frequency electromagnetic fields do not increase the rate of protein synthesis in human Skin fibroblasts as previously reported.
• Power-line frequency electromagnetic fields do not induce changes in phosphorylation, localization, or expression of the 27-kilodalton heat shock protein in human kera...
• Beta 2-adrenergic receptor activation delays dermal fibroblast-mediated contraction of collagen gels via a cAMP-dependent mechanism.
• Single Cell mechanics of keratinocyte Cells.
• Beta2-adrenergic receptor signaling mediates corneal epithelial wound repair.
• Vincent Azubike Ziboh (1929-2009).
• Hypoxic pre-conditioning increases the infiltration of endothelial Cells into scaffolds for dermal regeneration pre-seeded with mesenchymal stem Cells.
• Resveratrol Prevents High Fluence Red Light-Emitting Diode Reactive Oxygen Species-Mediated Photoinhibition of Human Skin Fibroblast Migration.

Schools

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• Albert Einstein College of Medicine of Yeshiva University

• Colum Presby

• New York University Med Center

• New York University School Of Med

Conditions Treated

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• Acanthosis Nigricans
• Acne
• Actinic Keratosis
• Athlete's Foot
• View All

Doctors Specialties

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• Dermatology

Accepted Insurances

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Education

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• NYU School of Medicine
• New York Presbyterian Hospital - Columbia Campus
• Albert Einstein College of Medicine of Yeshiva University

Hospital

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• University of California, Davis Medical Center

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