Associate Professor, Medicine, UCSD School of Medicine
Endocrinology
Dr. Seth J. Field, MD
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Seth Field is an expert in the field of Endocrinology. He attended Harvard Medical School. Harvard Medical School is a one of the best medicals schools. Harvard Medical School is ranked 1 in research and 12 in primary care medicine He received awards:"Elected Member". Seth Field has written publications. He has 31 publications published with PubMed. The lastest paper is: 'E2F-1 functions in mice to promote apoptosis and suppress proliferation.'
Publications
- GOLPH3 Links the Golgi, DNA Damage, and Cancer.
- DNA Damage Triggers Golgi Dispersal via DNA-PK and GOLPH3.
- GOLPH3L Antagonizes GOLPH3 to Determine Golgi Morphology.
- Role of phosphatidylinositol 4-phosphate (PI4P) and its binding protein GOLPH3 in HCV secretion.
- Receptor tyrosine kinases and TLR/IL1Rs unexpectedly activate myeloid cell PI3kγ, a single convergent point promoting tumor inflammation and progression.
- Regulation of oxysterol-binding protein Golgi localization through protein kinase D-mediated phosphorylation.
- GOLPH3 bridges phosphatidylinositol-4- phosphate and actomyosin to stretch and shape the Golgi to promote budding.
- Mutations in INPP5E, encoding inositol polyphosphate-5-phosphatase E, link phosphatidyl inositol signaling to the ciliopathies.
- E2F2 represses cell cycle regulators to maintain quiescence.
- Deletion of the phosphoinositide 3-kinase p110gamma gene attenuates murine atherosclerosis.
- The p85 regulatory subunit of phosphoinositide 3-kinase down-regulates IRS-1 signaling via the formation of a sequestration complex.
- A novel phosphatidylinositol(3,4,5)P3 pathway in fission yeast.
- Diabetes and exocrine pancreatic insufficiency in E2F1/E2F2 double-mutant mice.
- Identification and characterization of a phosphoinositide phosphate kinase homolog.
- The PHD finger of the chromatin-associated protein ING2 functions as a nuclear phosphoinositide receptor.
- Lipid research picks up speed on the slopes of Taos.
- The GOLPH3 pathway regulates Golgi shape and function and is activated by DNA damage.
- Multiplexed minipreps for rapid screening of large numbers of recombinant DNA clones.
- Myc requires distinct E2F activities to induce S phase and apoptosis.
- The E2F1-3 transcription factors are essential for cellular proliferation.
- Synthesis and activity of C11-modified wortmannin probes for PI3 kinase.
- E2F1 and E2F2 determine thresholds for antigen-induced T-cell proliferation and suppress tumorigenesis.
- The PX domains of p47phox and p40phox bind to lipid products of PI(3)K.
- E2F-1 functions in mice to promote apoptosis and suppress proliferation.
- A role for E2F1 in the induction of apoptosis during thymic negative selection.
- Accelerated DNA replication in E2F1- and E2F2-deficient macrophages leads to induction of the DNA damage response and p21(CIP1)-dependent senescence.
- Mutation of E2F2 in mice causes enhanced T lymphocyte proliferation, leading to the development of autoimmunity.
- Modification of protein sub-nuclear localization by synthetic phosphoinositides: evidence for nuclear phosphoinositide signaling mechanisms.
- PtdIns(4,5)P2 functions at the cleavage furrow during cytokinesis.
- Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance.
Schools
Harvard Medical School
Conditions Treated
- Abnormal Thyroid
- Hypothyroidism
Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- Elected Member
Education
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Massachusetts General Hospital -
University of Pennsylvania Health System -
Harvard Medical School
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