Krick-Brooks Chair in Nephrology, Professor of Medicine, Cancer Biology, Molecular Physiology and Biophysics, Vanderbilt University School of Medicine
Nephrology
Dr. Volker H. Haase, MD
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Volker Haase works in the field of Nephrology. Volker Haase is also published. He has 82 publications published. The lastest publication: "A lymphocyte-specific Ltk tyrosine kinase isoform is retained in the endoplasmic reticulum in association with calnexin.'
Publications
- Anaemia in Kidney disease: harnessing hypoxia responses for therapy.
- Endothelial HIF-2 mediates protection and recovery from ischemic Kidney injury.
- CD73-Dependent Generation of Adenosine and Endothelial Adora2b Signaling Attenuate Diabetic Nephropathy.
- Activation of hypoxia-inducible factor-2 in adipocytes results in pathological cardiac hypertrophy.
- A Breath of Fresh Air for Diabetic Nephropathy.
- Hypoxia-inducible factor regulates hepcidin via erythropoietin-induced erythropoiesis.
- Hypoxia-inducible factor signaling in the development of Kidney fibrosis.
- Mechanisms of Hypoxia Responses in Renal Tissue.
- Regulation of erythropoiesis by hypoxia-inducible factors.
- Hepatic HIF-2 regulates erythropoietic responses to hypoxia in Renal anemia.
- Endothelial HIF signaling regulates pulmonary fibrosis-associated pulmonary Hypertension.
- Hypoxia-inducible factor 2 regulates hepatic lipid metabolism.
- Molecular mechanisms of ischemic preconditioning in the Kidney.
- Hypoxia-inducible factor-2 (HIF-2) regulates hepatic erythropoietin in vivo.
- Inflammation and hypoxia in the Kidney: friends or foes?
- Muc1 is protective during Kidney ischemia-reperfusion injury.
- Proximal tubule sphingosine kinase-1 has a critical role in A1 adenosine receptor-mediated Renal protection from ischemia.
- Myeloid cell-derived hypoxia-inducible factor attenuates inflammation in unilateral ureteral obstruction-induced Kidney injury.
- Renal cancer: oxygen meets metabolism.
- Preischemic targeting of HIF prolyl hydroxylation inhibits fibrosis associated with Acute kidney injury.
- AT1A angiotensin receptors in the Renal proximal tubule regulate blood pressure.
- Angiotensin II: breathtaking in the Renal medulla.
- Epithelial Notch signaling regulates interstitial fibrosis development in the Kidneys of mice and humans.
- HO-1 in control of a self-eating Kidney.
- Sirtuins and their relevance to the Kidney.
- Renal oxygenation suppresses VHL loss-induced senescence that is caused by increased sensitivity to oxidative stress.
- Astrocyte hypoxic response is essential for pathological but not developmental angiogenesis of the retina.
- Hypoxic regulation of erythropoiesis and iron metabolism.
- The sweet side of HIF.
- Activation of sphingosine-1-phosphate 1 receptor in the proximal tubule protects against ischemia-reperfusion injury.
- Targeted deletion of Dicer from proximal tubules protects against Renal ischemia-reperfusion injury.
- VHL deletion impairs mammary alveologenesis but is not sufficient for mammary tumorigenesis.
- The glial cell response is an essential component of hypoxia-induced erythropoiesis in mice.
- Pathophysiological Consequences of HIF Activation: HIF as a modulator of fibrosis.
- Oxygen regulates epithelial-to-mesenchymal transition: insights into molecular mechanisms and relevance to Disease.
- Stable expression of HIF-1alpha in tubular epithelial cells promotes interstitial fibrosis.
- Hemoglobin in the Kidney: breaking with traditional dogma.
- Hypoxia-inducible factor augments experimental colitis through an MIF-dependent inflammatory signaling cascade.
- Hypoxia-inducible factor signaling in the development of tissue fibrosis.
- Epidermal sensing of oxygen is essential for systemic hypoxic response.
- Primary coenzyme Q deficiency in Pdss2 mutant mice causes isolated Renal disease.
- Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition.
- Hypoxia and podocyte-specific Vhlh deletion confer risk of glomerular Disease.
- Regulation of iron homeostasis by the hypoxia-inducible transcription factors (HIFs).
- The hypoxia-inducible factor alpha pathway couples angiogenesis to osteogenesis during skeletal development.
- Hypoxia-inducible factors in the Kidney.
- Suppression of Fas-FasL coexpression by erythropoietin mediates erythroblast expansion during the erythropoietic stress response in vivo.
- Cytoprotective effects of hypoxia against cisplatin-induced tubular cell apoptosis: involvement of mitochondrial inhibition and p53 suppression.
- pVHL function is essential for endothelial extracellular matrix deposition.
- Renal cyst development in mice With conditional inactivation of the von Hippel-Lindau tumor suppressor.
- Protection of HIF-1-deficient primary Renal tubular epithelial cells from hypoxia-induced cell death is glucose dependent.
- Hypoxia inducible factor 1 alpha regulates T cell receptor signal transduction.
- Decreased growth of Vhl-/- fibrosarcomas is associated With elevated levels of cyclin kinase inhibitors p21 and p27.
- Inactivation of the arylhydrocarbon receptor nuclear translocator (Arnt) suppresses von Hippel-Lindau Disease-associated vascular tumors in mice.
- Inflammatory hypoxia: role of hypoxia-inducible factor.
- Hypoxic induction of Ctgf is directly mediated by Hif-1.
- Epithelial hypoxia-inducible factor-1 is protective in murine experimental colitis.
- Vhlh gene deletion induces Hif-1-mediated cell death in thymocytes.
- DNA oligonucleotide microarray technology identifies fisp-12 among other potential fibrogenic genes following murine unilateral ureteral obstruction (UUO): modulation ...
- Hypoxia-induced gene expression occurs solely through the action of hypoxia-inducible factor 1alpha (HIF-1alpha): role of cytoplasmic trapping of HIF-2alpha.
- HIF-1alpha is essential for myeloid cell-mediated inflammation.
- Loss of vascular endothelial growth factor expression reduces vascularization, but not growth, of tumors lacking the Von Hippel-Lindau tumor suppressor gene.
- Hypoxia-inducible factor-2 regulates vascular tumorigenesis in mice.
- The VHL tumor suppressor and HIF: insights from genetic studies in mice.
- Low oxygen stimulates the immune system.
- The VHL/HIF oxygen-sensing pathway and its relevance to Kidney disease.
- Complete human NF1 cDNA sequence: two alternatively spliced mRNAs and absence of expression in a neuroblastoma line.
- Alternatively spliced ltk mRNA in neurons predicts a receptor With a larger putative extracellular domain.
- The murine NF2 homologue encodes a highly conserved merlin protein With alternative forms.
- Four tissue-specific mouse ltk mRNAs predict tyrosine kinases that differ upstream of their transmembrane segment.
- A lymphocyte-specific Ltk tyrosine kinase isoform is retained in the endoplasmic reticulum in association With calnexin.
- Vascular tumors in livers With targeted inactivation of the von Hippel-Lindau tumor suppressor.
- Ineffective erythropoiesis in Stat5a(-/-)5b(-/-) mice due to decreased survival of early erythroblasts.
- Deletion of Vhlh in chondrocytes reduces cell proliferation and increases matrix deposition during growth plate development.
- The VHL tumor suppressor in development and Disease: functional studies in mice by conditional gene targeting.
- The VHL tumor suppressor: master regulator of HIF.
- von Hippel Lindau tumor suppressor regulates hepatic glucose metabolism by controlling expression of glucose transporter 2 and glucose 6-phosphatase.
- Hypoxia activates the cyclooxygenase-2-prostaglandin E synthase axis.
- Loss of the tumor suppressor Vhlh leads to upregulation of Cxcr4 and rapidly progressive Glomerulonephritis in mice.
- Renal epithelium regulates erythropoiesis via HIF-dependent suppression of erythropoietin.
- The endothelial PHD2/HIF-2 axis regulates pulmonary artery pressure in mice.
Schools
JohannWolfgangGoetheUniversitaet Frankfurt Fachbereich Humanmedizin
Emory University
Massachusetts General Hospital
Beth Israel Hospital
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acute Kidney Failure
- Chronic Glomerulonephritis
- Chronic Kidney Diseases
- Diabetes With Renal Manifestations
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Doctors Specialties
- Nephrology
Accepted Insurances
Education
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Goethe University Frankfurt Medical Faculty -
Emory University -
Beth Israel Deaconess Medical Center -
Massachusetts General Hospital
Hospital
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Vanderbilt Hospital and Clinics
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